Junk DNA has long been an emotive issue. Some believe that much of our genome consists of useless genes while others believe that much of it serves a functional role.
Mainstream biologists do not subscribe to the belief that much of our genome consists of nonfunctional material; this claim has been popularised by Intelligent Design Creationists and others who promote this view.
Hypercommunication on the DNA level
Biologists have recently come to recognize that much of our human genome consists of non-coding DNA (the bits between genes). Although its genetic code cannot provide guidance to our cells on how to make proteins, this non-coding DNA no longer appears to be simply useless junk; rather it plays an essential role.
At its core, DNA may even play a fundamental role in our senses: intuition, hunches, premonitions, gut feelings, and synchronistic events. The non-coding parts of the genome can influence gene expression levels to affect how much protein a cell produces; hence why some people seem more prone than others to certain diseases.
Scientists have long sought ways to unlock the DNA’s hidden functions. Recently, scientists have discovered that “junk” DNA contains a system of switches which affect gene expression levels by turning up or down particular genes’ activity levels – like light bulbs with dimmers – influencing which genes are expressed by cells and whether or not they will become liver cells or neurons.
Researchers have recently made an astounding discovery: DNA encodes information that is passed along through electromagnetic and biophotonic waves known as bio-waves to other cells – known by some as hypercommunication. DNA can encode its language for these transmissions to other cells via these bio-waves.
This concept rests on the understanding that life is electromagnetic rather than chemical at every level of organismic, cellular-nucleotide, molecular and chromosomal holographic expression – from organismic through nucleotides, molecular and chromosomal-holographic communication with DNA using polarized bio-holographic signals containing both visual and textual information; external biowaves informed by endogenous DNA laser radiation can then manipulate these holograms for manipulation purposes.
Pjotr Garajajev and Vladimir Poponin, biophysicists who pioneered this research, demonstrated how living DNA could be altered with language-modified waves. By successfully reprogramming cells with new genomes – such as turning frog embryos into salamander embryos through vibration (sound frequencies) and language instead of cutting-out procedures as would normally be required – they demonstrated that living DNA could indeed be modified through simple vibration-based manipulations.
Inter-organism communication by the mean of invisible light
Scientists have long recognized that most genes originate from a common ancestor, copy themselves over evolutionary time, and evolve gradually until new genes emerge and join the complex network that regulates cell reactions. But there are exceptions: orphan genes – long stretches of DNA without matching protein sequences that have arisen independently in some remote part of nature.
Biologists have recently proposed an alternate explanation for the origins of orphan genes. They speculate that many may have arisen out of junk DNA – the non-coding genetic code at the core of our genomes – which scientists have studied by comparing fruit fly DNA sequences. As part of testing this theory, scientists compared orphan genes between species of fruit flies; orphan genes often shared short sequences with one or two other genes within that species but none from anywhere else within its genome, suggesting that non-coding DNA may mutate into functional genes over time resulting in novel protein-coding sequences being generated as a result.
This hypothesis has generated much discussion. Some have taken issue with it, claiming that the human genome project proved that most of our DNA is unnecessary and that no biochemical function exists for these regions of noncoding DNA. Furthermore, studies have indicated that junk DNA plays an integral part in producing ultra-weak light emissions called biophotons – raising further doubt about whether most of it may not serve any practical function at all.
Inter-organism communication through invisible light may sound farfetched at first, but in fact has been proven by experimental evidence in laboratories worldwide. Scientists have shown how lasers can be used to modulate DNA frequencies and alter its structure – this leads to biophoton emissions being altered accordingly and detected and analyzed afterwards.
Scientists are conducting experiments to see whether biophotons can also transmit information between cells. In one such experiment, a team of scientists used a laser to modulate junk DNA frequency before analyzing its biophoton emissions. They observed vibrational DNA molecules transmitted across their sample. This suggests junk DNA might play a significant role in transmitting information between cells; potentially providing us with clues into consciousness.
Hypermutation
Researchers from UC, Berkeley and Washington University recently unlocked an unanticipated function for one form of junk DNA. Transposons (ancient viral sequences capable of invading host genomes) proved essential to mouse viability; when deleted from mouse pups prior to birth. This marks the first time scientists have demonstrated that so-called junk DNA plays an essential role in gene regulation.
This discovery marks a breakthrough in understanding junk DNA’s function. To date, scientists had focused on understanding coding mutations such as deletions or insertions that alter amino acid sequences of proteins affecting their function; but without being able to pinpoint how mutations occurred.
Machine learning provides the key to unlocking these new functions of junk DNA, using artificial intelligence to predict how specific genes will respond to certain sequences of mutations, then compare those predictions against actual genomic sequence data to determine which mutations lead to which diseases.
Understanding how junk DNA can lead to disease is crucial as it may explain why there are so many mutations in the human genome. Most mutations don’t occur within genes coding for proteins but rather occur within regions of non-protein encoding regions of DNA known as junk DNA, which are frequently associated with disease and illness.
Scientists once believed that junk DNA came about via natural selection. Unfortunately, scientists were never able to establish exactly how much nonfunctional DNA exists in a genome or how fast its creation and deletion occurs; additionally it’s hard to see how extra DNA might actually benefit a species with large population sizes and fast population growth rates.
Begun made an important discovery in 2006 when he demonstrated that noncoding DNA can mutate into functioning genes through his comparison of gene sequences from Drosophila melanogaster with closely related species. Furthermore, Begun showed how different sequences from one genome can mutate into multiple functional genes over time, suggesting some junk DNA can become functional genes through mutation. His work led him to consider living systems chromosome continuums as waveform information realities.
Hyperreplication
Junk DNA refers to non-coding DNA found between our genome’s genes. Although much of this DNA remains mysterious, scientists have discovered that some forms contain important information regarding our cells’ functionality – suggesting that junk DNA contains not random sequences but instead contains short codes necessary for translating genes into functional proteins.
This hypothesis is founded on established genetic principles and observations made over decades, along with newer research into epigenetics and the cellular machinery responsible for transcription into RNA and protein. While the term junk DNA can be controversial, evidence supporting it is compelling; sequence conservation analyses consistently find that only 5-20% of the human genome is under detectable selective pressure whereas most of it consists of nonconserved junk DNA; additionally many transposable elements present within that 80-95% are rapidly decaying due to neutral mutational drift.
However, it remains elusive how such an enormous quantity of junk DNA could have amassed in the first place. Genes must be read and translated into functional proteins via transcription into RNA by regulatory components that indicate when and where genes should become active; sequences also must have start/stop short codes to signal when genes should begin or terminate operation.
No amount of DNA randomness could ever hope to achieve any of this. Generating genes out of such DNA would be like expecting Scrabble tiles to spell out coherent sentences on their own; mutations must accumulate to allow for its transcription into RNA, translated into protein and activated when and where needed.
Wang and his colleagues’ study suggests that some of the junk DNA in our genome contains important regulatory elements. They examined Alu transposable elements – which make up most of this noncoding junk DNA in human genome – as a family. When one gene from this family was knocked out in mice, half died before birth – further supporting this piece of noncoding DNA being essential to viability in mammals.
